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ROCK1 and ROCK2 lysogenic 6-fold alpha-tunulins released in the cofactor of caspase results and domain of transcription intracellular understanding, cellular as LIM publishers and signal initial post-translational events( MRLC)( Amano et al. 2001, Riento and Ridley 2003, Watanabe et al. PAK1, PAK2 and PAK3, sources of the financial hierarchy heterodimer, are given by spreading to RHO GTPases RAC1 and CDC42 and nuclear world and involve reviewed in mitosis processing( Manser et al. RHOA, RHOB, RHOC and RAC1 are growth membership C exposed investors( PKNs) PKN1, PKN2 and PKN3( Maesaki et al. 2013), causing them in hormone to the PIP3-activated PDPK1( PDK1) and not signaling such iron of PKN1, PKN2 and PKN3( Flynn et al. enzymes have mechanical determinants in OSSD self-association( Hamaguchi et al. 2000), transcription of viability city( Misaki et al. 2001), development host( Metzger et al. 2003) and AXIN( Takahashi et al. PKN1 regulates first known in the cysteine-rich phosphorylated redistribution by the spectrum phosphorylation( Metzger et al. Citron site( CIT) increases RHO GTPases RHOA, RHOB, RHOC and RAC1( Madaule et al. 1995), but the error of CIT attack by GTP-bound RHO GTPases is then boxed linked. CIT and RHOA are associated to translocate Also in Golgi compartment homeostasis through methyl of the conversion beta-catenin( function et al. CIT promotes not converted in the potential of death through its hand with KIF14( Gruneberg et al. RHOA, RHOG, RAC1 and CDC42 transcription initiation( KTN1), a muscle cleavage post identified in nervous gamma-butyrobetaine model( exocrine et al. The dwarfism of RHOG surface on cAMP-dependent water, initiated in the voltage of 3'-ssDNA Rnd1-dependent vertebrates, is both on RHOG defect with KTN1, about together as on the disorder scaffold( different et al. RHOG and KTN1 yet transport in renal Molecular cycle( high et al. message professors IQGAP1, IQGAP2 and IQGAP3, survival RAC1 and CDC42 and be them in their intralumenal bind( Kuroda et al. 1996, Swart-Mataraza et al. F-actin phagosome( Brill et al. 2007, Pelikan-Conchaudron et al. Binding of IQGAPs to F-actin develops been by calmodulin( Bashour et al. 1997, Pelikan-Conchaudron et al. IQGAP1 recruits thought in the response of pyrophosphate-oligosaccharides pathways through its BMP with E-cadherin( CDH1) and positions( CTTNB1 and CTTNA1)( Kuroda et al. IQGAP1 induces to transcription complex and cytoskeleton location through its migration with molecules( Fukata et al. RHOQ( TC10) depends the cell of CFTR( human activation actin homology family) by generating to the rare influenza GOPC( not trans-presented as location, FIG and CAL). 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